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Within this framework, our observation of reduced thalamic GSH during the interictal phase may be regarded as a candidate neurochemical correlate of these chronic, between-attack thalamic changes, possibly reflecting cumulative oxidative burden in a region chronically exposed to trigeminovascular input over years of recurrent attacks

Furthermore, LDL receptors become dysfunctional, increasing serum LDL-C, while inhibition of HDL-C concentration in the blood causes undesirable changes in lipid metabolism, producing hypercholesterolemia (Yousef et al

Marasinghe et al., 2005)